Posted: December 27th, 2014

Hematology 3.2

Hematology 3.2

Order Description

You are to create a concept map based on the given case study. You can either use the electronic version available on Dabbleboard, another computerized concept map

program, or you can draw your own. You may not copy one that is already done. Your submission must be your own original work.

Hematology 3.2
McCance & Huether Text:Chapters 25-28
Brashers Text: Case Studies # 12, #13
Concept Map Case Study
Within each lesson, one concept map is due. There are two parts. Make sure
you create the concept map as well as answer the critical thinking questions.
INSTRUCTIONS
You are to create a concept map based on the given case study. You can
either use the electronic version available on Dabbleboard, another
computerized concept map rogram,
or you can draw your own. You may not copy one that is already done. Your
submission must be your own original work.
Concept map requirements for inclusion
Demonstrate the pathophysiological concepts that pertain to this case study.
Indicate relationships between concepts —group clusters or patterns of signs
and symptoms
Complete the critical thinking questions
Demonstrate your understanding of the case by answering the critical
thinking
Questions
References should be in APA format.
CONCEPT MAP CASE STUDY ASSIGNMENT
Case:
E.J. is a 24 –year old teacher and avid mountain biker. She considers
herself very healthy as she exercises regularly (5 day/week aerobic
workouts; resistance training 3 sessions/week); follows a strict vegan
diet (she stopped eating all meat and dairy products ~ 3 years ago d/t
health and ethical reasons); doesn’t smoke and has no more than 1
glass of wine/meal ~ 4 times/week. She takes OCPs and a daily MVI.
She presents to your clinic with “no energy.”
Critical Thinking Question:
What are your recommendations for this patient for a management
plan, given the diagnosis?

A diagram demonstrating the pathophysiology of hypoxia and cellular injury
an eaample of decrease blood flow
decreasei‘cessation of blood flow ._”) would be blocked vessel of the
heart
i oxidative phosphorylation is the
mechanism within the cell which
lack of oxygen will result with a fall of oxidative phosphorylation -“”) transfers the mm from substrates to
ATP
ATP not only stores energy but i
also transfers it from one
this? Am,“ pugp is respfins‘bii molecule to another (ATP is used
Hf toting?” 01?“ 9mm“ ( depletion of cellular ATP / the role of mitochondria wihtin the cell is cellular
a 3 a g g
acidis agaisnt the concentration gradient; w m) will result of mitochondrial swelling
thus allowin the trans nation of these
ions g po 1 phosphorylation that generates most of the cell’s ATP
failure of membrane Nail failure of membrane
which can to ATPZSC CZlClum pumps SWitch io anaemblc metabolism when the cell lacks oxygenl
electrical excitability of the I I $ attempt to work in an anaerobic
nerve and muscle cells ‘ a t boli i ‘th t ‘ v
K influx om. & mm; of ‘L me a c manner (M on oxygen)
efflux 0”” mo 1m synthesis depletion of glycogen
I I mcreaed lactic acid
swelling of endoplamic fan in intracellular pH
an example of electrical excitability would reticulum hos holi ases
be; a patient experiencing ventricular I
tachycardia as seen on a heart monitor
plasma membrane damage – liberation & activation of lysosomal enzymes

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