Posted: May 23rd, 2017
The primary biochemical lesion in homozygote with familial hypercholesterolemia (type IIa) is
The primary biochemical lesion in homozygote with familial hypercholesterolemia (type IIa) is (A) Loss of feed back inhibition of HMG reductase (B) Loss of apolipoprotein B (C) Increased production of LDL from VLDL (D) Functional deficiency of plasma membrane receptors for LDL
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